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August 24, 2019, 05:35:06 am

Author Topic: VCE Biology Question Thread  (Read 1309406 times)  Share 

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vox nihili

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Re: VCE Biology Question Thread
« Reply #6870 on: October 29, 2015, 04:58:11 pm »
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Could someone please elaborate on this question, I was stuck between b and c. Thanks

This question still gives me nightmares  :'(
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BakedDwarf

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Re: VCE Biology Question Thread
« Reply #6871 on: October 29, 2015, 05:12:57 pm »
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Each pyruvate in the Krebs cycle is broken down to form 3 carbon dioxide molecules, 1 ATP and 5 loader acceptor molecules... right?

Would I get a mark for this definition of pathogen?
Pathogen is a cellular or non-cellular disease-causing agent

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Re: VCE Biology Question Thread
« Reply #6872 on: October 29, 2015, 05:17:41 pm »
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http://i.imgur.com/VK9S9ET.gif

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Biology24123

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Re: VCE Biology Question Thread
« Reply #6873 on: October 29, 2015, 05:26:13 pm »
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How much should we know about apoptosis? Is this enough information

Signal molecule binds to death receptor on the cell membrane
This triggers a signal transduction pathway with the activation of caspase proteins
Caspases cleave certain parts of the cell which causes blebbing


THEBEAST

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Re: VCE Biology Question Thread
« Reply #6874 on: October 29, 2015, 05:41:02 pm »
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Can someone please explain the role of the lymphatic system/lymph nodes in immunity?

cosine

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Re: VCE Biology Question Thread
« Reply #6875 on: October 29, 2015, 05:54:49 pm »
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Can someone please explain the role of the lymphatic system/lymph nodes in immunity?

Lymph nodes and lymphatic system are not the same thing, lymphatic system is the whole system, the collection of the organs/tissues that are responsible for the lymphatic processes/cells.

Lymph nodes are bean-like structures/organs? that are found throughout the body. They trap foreign pathogens and store lymphocytes in them. So B cell proliferation occurs in the lymph nodes, so when you get an infection that is usually why your lymph nodes enlarge, because B cells are proliferating rapidly.
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BakedDwarf

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Re: VCE Biology Question Thread
« Reply #6876 on: October 29, 2015, 05:58:58 pm »
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How much should we know about apoptosis? Is this enough information

Signal molecule binds to death receptor on the cell membrane
This triggers a signal transduction pathway with the activation of caspase proteins
Caspases cleave certain parts of the cell which causes blebbing

Apoptosis is a genetically controlled, programmed cell death that can be initiated by internal and external signals.
It occurs in:
- healthy cells that are no longer needed
- damaged cells and those no longer functioning normally
- immune cells that damage self cells

You should be able to distinguish it from necrosis, which is a non-programmed death of a cell that releases dangerous chemicals into its surroundings and harms neighbouring cells.

I don't think we need to know how apoptosis is initiated.

Can someone please explain the role of the lymphatic system/lymph nodes in immunity?

Lymph nodes are small masses of lymph tissue that are dense with white blood cells, and are found along lymph vessels
(basically what cosine said)

winchesteraddict

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Re: VCE Biology Question Thread
« Reply #6877 on: October 29, 2015, 07:06:35 pm »
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Could someone please elaborate on this question, I was stuck between b and c. Thanks
T-Rav is correct, this question is pretty weird.
However, imo the closest correct answer would be (b) Phagocytes.
I'm guessing that this is an inflammatory response. Seratonin (released by mast cells, philosophy etc) has already increased the permeability of the vessels to allow phagocytes to move into the area. Early phagocytes would of-course release histamine to attract more phagocytes to the area (could also be d). Phagocytes would then attack the bacteria, therefore closest one could be (b).

However I might be wrong :)

warya

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Re: VCE Biology Question Thread
« Reply #6878 on: October 29, 2015, 07:09:51 pm »
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So clonal selection/expansion occurs at the lymph nodes
And memory b/t cells reside/routinely scan the lymph fluid, so upon encounter with specific antigen, they produce a secondary response right?

And NK cells kill any cell displaying non self antigens or displaying non self antigens on MHC1?
And cytotoxic T cells kill infected body cells displaying a specific antigen on MHC1?

Is all this correct?
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Jay.C

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Re: VCE Biology Question Thread
« Reply #6879 on: October 29, 2015, 07:55:08 pm »
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someone pleaseeeeeeee show me how to do a dihybrid cross between linked genes!!!
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BakedDwarf

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Re: VCE Biology Question Thread
« Reply #6880 on: October 29, 2015, 08:07:25 pm »
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someone pleaseeeeeeee show me how to do a dihybrid cross between linked genes!!!

Is this what you want?

Jay.C

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Re: VCE Biology Question Thread
« Reply #6881 on: October 29, 2015, 08:28:00 pm »
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Is this what you want?

yes!!!! thanks heaps, lol didnt think it was going to be that simple. thanks again!
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StupidProdigy

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Re: VCE Biology Question Thread
« Reply #6882 on: October 29, 2015, 09:00:27 pm »
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Is this what you want?
What about recombinants? Do we ignore them usually or?

How can a signal transduction pathway results in many responses?
And also, what are two factors that affect the rate of change of dna? (dunno if this just wants mutagens or not..) thanks
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cosine

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Re: VCE Biology Question Thread
« Reply #6883 on: October 29, 2015, 09:42:50 pm »
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What about recombinants? Do we ignore them usually or?

How can a signal transduction pathway results in many responses?
And also, what are two factors that affect the rate of change of dna? (dunno if this just wants mutagens or not..) thanks

The second messenger (in the case of hydrophillic hormones) are activated upon the hormone-complex formation. This activated second messenger then triggers other intracellular biochemical pathways/reactions and results in a cascade of reactions, eventually leading to a specific cellular response.

Two factors:
- Radiation
- UV light? Idk about this one but i think they want mutagens
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cosine

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Re: VCE Biology Question Thread
« Reply #6884 on: October 29, 2015, 10:15:30 pm »
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Urgent help!

In the 2013 exam the last question about the embryonic thing, you know how the question asks about the UV light, the answer says that it just destroys the nucleus? How does it do this? I thought UV light would make the nucleus visible so it could be destroyed, is this what the answer means?
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