Hey everyone,
I was just wondering whether anyone knew where I could find some resources such as practise exams or practice questions maybe even videos that could help me ace my Unit 2 Biology exam. I know they could be really rare due to it being a year 11 subject but I still want to do well on it. Hopefully, someone could help me out? Thanks, everyone!
Hey!
I found the Amoeba sisters youtube channel to be quite helpful to understand concepts as they explain it in a very entertaining, funny but also educational way! Also, they have practice questions and worksheets on their website for you to consolidate your knowledge.
Another resource I would suggest is KhanAcademy. For me, doing their practice questions helped and also exposed me to some content I didn't know before. I used this resource for my first SAC.
From a quick google, I found this resource as well
https://mrlewis-vcebiology.weebly.com/unit-2.html where there are files you can download with powerpoints. Might be helpful.
Also, do you have textbook for biology? If you do, it would be worth doing questions from the chapter reviews and marking them yourself or asking a teacher to mark it.
The Biology 1 2 Checkpoints is also good to do because they have relevant questions that have popped up in exams.
This was a random list I made from the top of my head. I hope this helps!
Hi, I don't understand the answer to q3bii from the 2017 bio exam, was wondering if anyone could help? Much appreciated
So, you know that Lovastin is a competitive inhibitor. Here, the HMG-CoA is the real substrate that binds to the HMG-CoA Reductase enzyme where they have a complementary shape to each other. When the HMG-CoA substrate binds to the enzyme, there is an increase in blood cholesterol.
So, you want there to be a higher concentration of Lovastin (the competitive inhibitor) so that it is more likely for the Lovastin to bind to the HMG Reductase enzyme due to successful collisions, rather than the normal HMG-CoA substrate from binding so that the cholesterol cannot be produced, thus resulting in a lower concentration of cholesterol.
You don't want the Lovastin concentration to be too low because it would be more likely for the normal HMG substrate to bind to the enzyme, resulting in cholesterol being produced and high cholesterol and we do not want that.
I hope this makes sense!